By Frank J. Dixon (Eds.)
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Additional resources for Advances in Immunology, Vol. 47
123, 918. , and Yamamura, Y. J. Immunol. 119,159. , Urban, J. , and Ishizaka, K. J. Zmmunol. 121, 2413. , and Ishizaka, K. J. Immunol. 125,148. , and Kishimot, T. J. Immunol. 127, 465. , and Kishimoto, T. J. Immunol. 130, 1056. , and Okumura, K. (1971). J. Immunol. 106, 1012. , and Ishizaka, K. (1975). Cell. Zmmunol. 20, 276. ,and Ishizaka, K. J. Immunol. 116, 1257. , and Ishizaka, K. J. Immunol. 117, 1211. Thorley-Lawson, D. A , , Nadler, L. , Bhan, H. , and Schooley, R. T. (1985). J. Immunol.
1980) reported that antiidiotypic antibody suppressed primary IgE antibody response and ongoing antibody formation against the benzyl penicilloyl group. They also demonstrated in BALB/c mice that the production of antiidiotypic antibodies against syngeneic anti-OVA antibodies resulted in depression of both antihapten and anticarrier IgE antibody formation against DNP-OVA (Blaser et a l . , 1981). The third approach is to regulate the IgE antibody response by inducing antigen-specificsuppressor T cells.
1985). , 198413). It appears that GEF induces the activation of phospholipase A2 and phospholipase C in the T cells. Since GIF is a derivative of phospholipase-inhibitory protein, antagonistic effects between GEF and GIF with respect to the glycosylation of IgE-BFs may be related to activation versus inactivation of phospholipase A2 in the cells by the two lymphokines. REGULATION OF IgE BIOSYNTHESIS 27 3. Formation of IgE-BFs by Antigen-Specijic T Cells Induction of IgE-PF or IgE-SF formation by lymphokines, described above (cf.
Advances in Immunology, Vol. 47 by Frank J. Dixon (Eds.)